Low-dust joint compounds causes particles to bind together during sanding operations.
Hazard Analysis — Construction dust
Workers who tape and sand wallboard may be exposed to construction dust.
Chronic obstructive pulmonary disease (COPD) includes chronic bronchitis and emphysema. Chronic bronchitis is present when someone has a regular cough with phlegm for at least 3 months per year for two years. Emphysema is present when there is destruction of the walls of the airspaces of the lung. The destruction of airspaces means there is less lung surface; loss of lung surface reduced the ability of the lung to transfer oxygen into the blood stream.
Chronic obstructive pulmonary disease (COPD) includes chronic bronchitis and emphysema. Chronic bronchitis is present when someone has a regular cough with phlegm for at least 3 months per year for two years. Emphysema is present when there is destruction of the walls of the airspaces of the lung. The destruction of airspaces means there is less lung surface; loss of lung surface reduces the ability of the lung to transfer oxygen into the blood stream.
In 1999 COPD ranked as the fourth leading cause of death with over 124,000 deaths, and was a primary or contributing cause of death for an estimated 8.5% of the U.S. population. An estimated 16 million individuals in the U.S. had a diagnosis of COPD in 1994. Worldwide, COPD is projected to be the third leading cause of death by 2020. Eighty to ninety percent of COPD is caused, at least in part, by tobacco smoking, but exposure to dust is an important risk factor as well. The American Thoracic Society concluded that 15% of COPD was caused by occupational exposures, and that occupational dust and fume exposures can cause clinical bronchitis and loss of lung function in both smokers and nonsmokers. Dust may contain specific contaminants such as crystalline silica or hexavalent Chromium.
Smoking is the primary cause of COPD, but smoking alone does not explain all COPD, as only 15-20% of smokers developed COPD and 10% of deaths from COPD occur in persons who never smoked. Occupational exposures to dusts and chemicals (vapors, irritants, fumes) also cause COPD; the specific exposures in construction linked to COPD are wood dusts, cadmium, silica, welding fumes, cement dust, asbestos, and possibly isocyanates. For someone with heavy dust exposure over many years, the dust contributes as much to COPD as smoking does. Across all occupations, about 15% of COPD is caused by occupational exposures.
One study, “A case-control study of airways obstruction among construction workers”, examined occupational exposures to vapors, gases, dusts and fumes (VGDF) among older construction workers between 1997 and 2013, comparing 834 workers with COPD and 1,243 controls. Approximately 18% (95% CI=2-24%) of COPD in this population can be attributed to workplace exposures associated with construction tasks. After adjusting for smoking habits in this population, the study also revealed among construction workers who never smoked, 32% (95% CI=6-42%) of COPD was attributable to the workplace.
Airflow in nonsmokers without respiratory disease declines by 25 to 30 ml per year beginning at about age 35. The rate of decline of airflow is faster for smokers than for nonsmokers, and faster for those exposed to lots of dust. Individuals with COPD have more frequent chest illnesses, which also decrease lung function for several months.
When wood dust comes into contact with the skin, its non-allergic effect is usually irritation, which can lead to rashes or irritant dermatitis that tends to appear on the scalp, eyelids, neck, forearms, the backs of hands and genitals. These symptoms usually take approximately 15 days to develop and will only persist as long as the affected skin site remains in contact with the source of the wood dust.
Wood dust can also cause allergic contact dermatitis which is another form of dermatitis caused by sensitizers of certain wood dust species. This type of allergic reaction can produce the same effects as the non-allergic reaction to wood dust. The difference is that once the body is sensitized, it will set up an allergic reaction that can enable the skin to react more severely to even a small amount of that particular wood dust (or related wood species) in subsequent episodes of exposures.
Exposures to wood dust can affect the respiratory tract, such as the nose. Inhalation of wood dust can cause runny nose (rhinitis), sneezing, stuffed nose and nose bleeds. These symptoms usually arise from irritation, but can also be a consequence of allergic sensitation (i.e. rhinitis). Several studies have suggested that certain wood dusts, especially oak and beech (confirmed human carcinogens), but perhaps also birch, mahogany, teak, walnut (suspected human carcinogens) may cause nasal cancer, although rare.
Inhalation of wood dust can lead to obstructive patterns of respiratory changes, which can limit breathing flow rates and impair lung function. One lung condition attributable to wood dust exposure (especially Western Red Cedar) is occupational asthma, which includes wheezing, coughing, tightness across the chest and shortness of breath. Some wood dust can also cause asthma as an allergic reaction once the body is sensitized to that wood species.
Wood dust that comes into contact with the eyes can cause watering, soreness and inflammation of the eyes (conjunctivitis).
Level of Risk:
In 1999 COPD ranked as the fourth leading cause of death with over 124,000 deaths, and was a primary or contributing cause of death for an estimated 8.5% of the U.S. population. An estimated 16 million individuals in the U.S. had a diagnosis of COPD in 1994. Worldwide, COPD is projected to be the third leading cause of death by 2020. 80 to 90% of COPD is caused, at least in part, by tobacco smoking, but exposure to dust is an important risk as well. The American Thoracic Society concluded that 15% of COPD was caused by occupational exposures, and that occupational dust and fume exposures can cause clinical bronchitis and loss of lung function in both smokers and nonsmokers. Dust may also contain specific contaminants such as crystalline silica or hexavalent Chromium.
Most construction workers are at risk of COPD; studies find an elevated prevalence of COPD across many trades, and most trades have tasks that create dust or fumes. Many construction tasks have exposures to levels of dust which exceed the OSHA permissible exposure limit of 5 mg/m3 for non-specific dust. Because many construction trades work alongside each other, a construction worker can be over-exposed to dust generated by a co-worker doing a different task.
Some experts have suggested that dust at construction sites is best described as quartz (silica) containing mixed dust, and quartz is clearly one component of dust related to lung injury. The proportion of quartz in the overall dust exposure of any individual will vary by task, materials, specific years worked, work location, and likely other factors as well. Tuckpointing had respirable dust levels as high as 8 mg/m3 as an 8 hour TWA, with quartz levels as high as 1.7 mg/m3 as an 8 hour TWA. In one study that looked at both exposure and disease, workers with higher cumulative dust exposures were more likely to have a chest x-ray showing mixed dust pneumoconiosis (lung scarring due to an exposure to mixed dust). The prevalence of mixed dust pneumoconiosis was significantly increased after 25 years work at a tuckpointer or 33 years work as a concrete driller or grinder.
Studies of miners have shown that long term exposure to dust at levels of 2 mg/m3 causes COPD, and that as the silica content of the dust increases the total exposure needed to cause disease decreases. However, other dusts and fumes, including asbestos, cadmium and welding fumes also cause COPD, so silica is only one of the injurious components of construction dust.
Bergdahl IA, Toren K, Erikson K, Hedlund U, Nilsson T, Flodin R, Jarvholm B. Increased Mortality in COPD among construction workers exposed to inorganic dust. Eur Respir J 2004. 402-406
Exposure to Dust in Drywall Finishing:
Aubrey K. Miller, et. al., Health Hazard Evaluation Report on Control of Drywall Sanding Dust Exposures. NIOSH HETA 94-0078-2660 (1997).
Epling C, etal. Airborne Exposures and Ambulatory Peak Expiratory Flow in Drywall Finishers. (1999); viewed 07/22/09.
Methner MM. Identification of Potential Hazards Associated with New Residential Construction. Applied Occup Envir Hygiene. 15(2): 189-192; 2000.
NIOSH. Control of Drywall Sanding Dust Exposures. Appl Occupat Envir Hygiene 15(11): 820-821; 2000.
Verma DK, Middleton CG. Occupational Exposure to Asbestos in the Drywall Taping Process. Am Indust Hyg Assoc Journal 41(4): 264-269; 1980.
Young-Corbett, Deborah E. and Nussbaum, Maury A. Dust Control Effectiveness of Drywall Sanding Tools. JOEH 6 (7): 385- 389: 2009.
Young-Corbett, Deborah E. and Nussbaum, Maury A. Dust Control Technology Usage Patterns in the Drywall Finishing Industry. JOEH 6 (6): 315-323: 2009.
You must determine whether exposures at your jobs exceed allowable limits for overall dust and specific dusts.
Silica is an important component of many construction dust exposures., and the PEL for silica exposure in construction is much lower than the PEL for non-silica containing dust, so it is essential to determine the silica content of any dust exposures. OSHA has an e-tools site for silica that walks you through the process of assessment of silica exposure.
Exposure assessment must be done by a qualified person. You may be able to to obtain assistance in measuring exposures from your State OSHA consultation service or from your contractor association.
Regulations & Standards:
OSHA standard 1926.55, titled Gases, vapors, fumes, dusts, and mists, in safety and health regulations for construction must be in place. Exposure of employees to inhalation, ingestion, skin absorption, or contact with any material or substance at a concentration above those specified in the "Threshold Limit Values of Airborne Contaminants for 1970" of the American Conference of Governmental Industrial Hygienists, shall be avoided.
The current OSHA standard for overall dust which contain < 1% silica is 15 mg/m3. The NIOSH recommended exposure limit for crystalline silica is 0.05 mg/m3. NIOSH research shows that controlling exposures below this concentration should prevent all workers from obtaining silicosis. American Conference of Governmental Industrial Hygienists (ACGIH) recommends a time weighted average exposures of less than 10 mg/m3 for total dust. For respirable dust the ACGIH recommends a limit of 3 mg/m3 compared to an OSHA PEL for general industry of 5 mg/m3.
Federal OSHA Standards are enforced by the U.S. Department of Labor in 26 states. There are currently 22 states and jurisdictions operating complete State plans (covering both the private sector and state and local government employees) and 5 - Connecticut, Illinois, New Jersey, New York and the Virgin Islands - which cover public employees only. If you are working in one of those states or jurisdictions you should ensure that you are complying with their requirements.
The powered wall and ceiling sander with a vacuum attachment is used to flush drywall joints, seams, and screw holes after mud has dried, and smooth and prepare paint and other surfaces. The vacuum attachment collects dust product by the tool at its source. The pole or handle helps position the sander at an optimal position for the worker.
BIM is a concept that offers software application to integrate building information for hazard identification and safety planning.
A software program that lets users match video feed of work processes, or tasks being performed, with real-time exposure measurements.
Personal Protective Equipment
A respiratory protection program addresses exposures to airborne dust, engineered nanomaterials, and chemical exposures as well as implementing and evaluating other effective exposure control measures.